In hypopituitarism, this congenital disease is a result of the lack of growth hormone secreted by the anterior pituitary gland.
Pituitary dwarfism (normal proportion).
In a primary disorder or hypothyroidism, due to a thyroid gland problem, these are the lab findings for TH (T3 and T4 and TSH).
Low TH (T3 and T4) and increased TSH.
Increased serum calcium and PTH, and low serum phosphorus are lab findings for this parathyroid disorder.
Hyperparathyroidism.
This inhibits the release of ADH, resulting in an increase in urine production and dehydration when consumed.
Alcohol
Excessive epinephrine and norepinephrine production in pheochromocytoma result in these key signs and symptoms.
Hypertension, tachycardia, hyperglycemia, vomiting, severe headache, apprehension, palpitations.
This is kept at bedside for adult patients with hyperpituitarism due to dysphasia from tongue enlargement.
Tacheostomy kit.
Key nursing considerations for Levothyroxine, a thyroid replacement oral drug used for treatment of hypothyroidism, and a key side effect to monitor for.
Take it in the morning on an empty stomach, 1 hour before meals. Calcium, iron supplements, cholesterol drugs, and antacids interfere with absorption - take 4 hours apart. Monitor for S/S of hyperthyroidism.
The diet recommended to a patient diagnosed with hypoparathyroidism.
High calcium, and low phosphorous foods.
Hypertonic or 3% fluids are used in treatment of SIADH to encourage the shift of this electrolyte.
Sodium (correct the hyponatremia)
These are key signs and symptoms and lab findings of Cushing’s syndrome, from excess adrenocorticoids (especially cortisol).
Buffalo hump, purple striae on the abdomen, moon face, hirsutism, osteoporosis. Hypoglycemia, hypernatremia, and hypokalemia.
These are two injections to treat growth hormone secretion disorders, a GH inhibitor and a GH analog.
Ocreotide (inhbitor) and Somatotropin (analog).
Heat intolerance, excessive sweating, weight loss despite increased appetite, and dysrhythmias are symptoms of what thyroid disorder.
Hyperthyroidism.
BACKME, signs and symptoms of hypercalcemia found in patients with hyperparathyroidism.
Bone pain, arrhythmias, cardiac arrest, kidney stones, muscle weakness, excessive urination.
Diabetes Insipidus patients have diluted urine due to severe dehydration, resulting in this lab finding regarding specific gravity.
Specific gravity < 1.005
This drug class is used for Addison‘s disease, and s a lifelong drug that you increase during periods of stress, and take 2/3 in the morning and 1/3 in the afternoon.
Corticosteroids.
This is the most common surgical approach in a hypophysectomy, through the floor of the nose and the sphenoid sinuses, because the anterior pituitary gland is located here.
Trannsphenoidal, Sella Turcica.
This drug class blocks TH conversion, and is used during a Thyroid Storm, in additional to this non-specific Beta-Blocker.
Corticosteroids, and Propanolol.
Strider, wheezing, result from this airway complication, requiring this at bedside to create an airway and a medication to reverse hypocalcemia.
Layngospasm, tracheostomy set and injectable calcium gluconate.
Lifelong hormone replacement therapy is required for which disorder related to ADH.
Diabetes Insipidus.
These are some possible precipitating factors of an addisonian crisis, putting a patient at risk for vascular shock and death.
Stress, infection, dehydration, emotional turmoil, strenuous activity, medication inadhearance, iatrogenic (surgery), or rapid withdrawal of steroids.
These are 3 target organ deficiencies to monitor for post hypophysectomy.
Adrenal insufficiency, hypothyroidism, Diabetes Insipidus.
Thyroidectomy post-op care consists of monitoring for tetany, due to this electrolyte imbalance from possible accidental parathyroid gland removal.
Hypocalcemia.
Parahormone and calcitonin are antagonists, which affects this serum electrolyte in parathyroid disorders.
Differing lab values on ADH depending on whether the diabetes insipidus is neurogenic or nephrogenic.
Neurogenic = decreased ADH. Nephrogenic = elevated ADH.
Excess aldosterone being relased from the adrenal cortex in Conn’s syndrome, results in these electrolyte imbalances, which is treated with this specific diuretic.
Hypokalemia and hypernatremia. Spironolactone.