Sepsis I
According to Cecil's reaching this body temperature may be diagnostic of Sepsis.
Fever (>38.3° C)
Hypothermia (core temperature <36° C)
leading cause of morbidity and death among hospitalized patients.
SEPSIS
Meningitis is defined and identified by...
inflammation of the leptomeninges that cover the brain and spinal cord. It is identified by an abnormal increase in the number of white blood cells in cerebrospinal fluid (CSF).
Diagnosis of cavernous sinus thrombosis is usually made by this imaging modality.
MRI with a venogram
seventh planet from the sun
Uranus
3 possible inflammatory variables found in sepsis.
1. Leukocytosis (WBC count >12,000 mm 3 )
2. Leukopenia (WBC count <4000 mm 3 )
3. Normal WBC count with more than 10% immature forms
4. Plasma C-reactive protein >2 SD above the normal value
5. Plasma procalcitonin >2 SD above the normal value
this is the rate of sepsis mortality
20% - 30%
The leading causes of bacterial meningitis
S. pneumoniae (58% of cases),Streptococcus agalactiae (18% of cases), Neisseria meningitidis (14% of cases), H. influenzae (7% of cases), andListeria monocytogenes (3% of cases).
Septic thrombosis of the lateral sinus results from acute or chronic infections of this structure.
the middle ear. The infection spreads through emissary veins that connect the mastoid with the lateral venous sinus.
Bang! Pow! Sizzle! are examples of this.
onomatopoeia
4 groups that are most susceptible to Sepsis.
The rates are highest for premature infants, the advanced elderly (especially those older than 85 years of age), and patients with intravenous catheters, implanted devices, or severe medical morbidities such as severe burns or hematologic malignancies.
2 groups you may not see a fever response even though Sepsis is highly suspected.
Many patients have fever or chills, but older patients and those on immunomodulating medications may not mount a fever.
Because the size of the intravertebral canal remains relatively constant but the circumference of the spinal cord changes, spinal abscess formation is maximal in these regions.
thoracic and lumbar regions and minimal at the cervical spine enlargement.
Septic cavernous sinus thrombosis usually results from spread of infection through facial veins or from these facial structures.
the sphenoid or ethmoid sinuses
This Chess piece is only allowed to move diagonally.
A bishop.
Those with Diabetes would be most susceptible to a Sepsis infection from these organisms.
Mucormycosis, Pseudomonas species, Escherichia coli, group B streptococci
Tachypnea may be an indicator of respiratory compensation for these two conditions.
underlying metabolic acidosis or the early signs and symptoms of ARDS.
The pathologic hallmarks of CJD.
spongiform or vacuolar changes in the brain without cellular inflammatory infiltrates.
Neurologic Complications of Infective Endocarditis are most often due to this...
Most complications are related to valvular vegetations. Cerebral (but not systemic) emboli from mitral valve endocarditis are increasingly common. Most emboli, regardless of the bacterial cause of the infection, occur before or early in the course of treatment.
This color is at the top of a Rainbow.
Red
Septic shock is initiated by systemic release of an array of vasoactive mediators, including mostly this molecule.
nitric oxide (NO) produced by cytokine-inducible NO synthase (iNOS).
Bacteremia caused by several gram-negative organisms, including P. aeruginosa and enteric organisms, can result in this serious skin condition.
ecthyma gangrenosum, particularly in immunocompromised patients.
The clinical tetrad supporting the diagnosis of CJD.
subacute progressive dementia, myoclonus, typical periodic complexes on electroencephalography, and normal CSF
This is the Treatment for CJD.
There is no TREATMENT!!!!
Vexillology studies this.
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