subconscious clenching of the fist over the chest
levine sign
this ion imbalance prolongs qt
hypocalcemia
this is happening during the PRI
av node pauses with the stimulus and ventricles fill with blood
irregular rhythm, rate of 70, more p waves than qrs, pri increases in length before dropping qrs, narrow qrs
2nd degree type 1
1st intervention when no pulse
cpr
associated symptoms other than chest discomfort
anginal equivalents
this ion starts depolarization
sodium (by rushing in to cell)
20-40. 40-60. 60-100
intrinsic rates of purkinje system, AV node, SA node respectively
regular rhythm, rate of 30, no p waves, no pri, wide qrs
idioventricular
this medication and dose is indicated for torsades de pointes
magnesium sulfate 1-2 grams
plaque buildup within the walls of blood vessels which leads to partial or complete blockage of blood flow
atherosclerosis
this ion maintains the mechanical contraction/depolarization
calcium
stimulation of this slows SA node discharge and AV conduction
vagus nerve
regular or irregular rhythm, rate of 90, no p waves but f waves, pri is indiscernable, qrs present
aflutter
hyperventilation
series of cardiac conditions caused by an abrupt reduction in coronary artery blood flow
ACS
the movement of ions for the sodium potassium pump
3 Na out, 2 K in
this wave represents hypothermia
J wave (osborn)
regular rhythm, rate of 75, no p waves, no PRI, narrow qrs
Accelerated junctional
an intervention performed after ROSC when the pt. is still comatose
TTM
abnormal conduction through the ventricles
aberrant
this electrolyte state causes increased myocardial irritability
hypokalemia
patients have an accessory pathway that bypasses the AV node and causes early ventricular depolarization
WPW
these two findings suggest PE
RBBB. S1Q3T3 pattern
These are the H's and T's
Hypovolemia Hypoxia Hydrogen ions Hypo/Hyperkalemia Hypothermia
Tension pneumo Tamponade Toxins Thrombosis (P,T)