Antibodies
Involved in clumping action
Ig M
lymphocyte mature into T cells by passing through this body structure
thymus
spirochetes use these structures
tapered ends
bind to iron making it unavailable to invading pathogens
blood transferrins
set of rules used to establish a solid connection between pathogen and set of signs and symptoms
Koch's postulates
First responder to new infections
Ig M
reaction to a TB test or organ transplant
TD cells
enzyme used to produce clots
coagulase
sticky surface on skin keeps trapped invaders in place
sebum
this pathogen was an exception to Koch's postulates until serum media was used
mycoplasma
Dimer structure
Ig A
Destroys infected host cells
Tc cells
Viruses and parasitic protozoa use this to change its surface
antigenic variation
chemical attraction of a phagocyte to an invader
chemotaxis
Pure culture from diseased or dead animal is injected into healthy animal of the same species to produce same s/s from diseased animal
postulate 3
High levels in blood serum indicate parasitic worm infection or allergies
Ig E
sends signal to Tc cells to destroy a target cell
TH cell
protein produced by Neisseria gonorrhoeae for attachment
opa protein
complement proteins create holes in a pathogen's cell membrane
membrane attack complex
signs and symptoms from a suspected pathogen must be the same in every case of the disease
postulate 1
High antibody titer upon 2nd exposure
Ig G
activates T-dependent B cells to produce antibodies
TH cells
protein produced by Streptococcus pyogenes for attachment
M protein
type of cytokine produced during a viral infection
interferons
infected/diseased animal sample is cultured in a petri dish in lab setting and is observed under a microscope
postulate 2