All About DKA
HHS Happenings
Don't Stress... Assess!
Beyond the A1C
Concept Cousin's (They're related!)
100

This primary hormone is deficient or absent in DKA, leading to uncontrolled glucose.

Insulin

100

This type of diabetes is most commonly associated with HHS.

Type 2 Diabetes

100

The medical term for high blood sugar

Hyperglycemia

100

This laboratory test will reveal the presence of ketones and glucose in the urine. 

Urinalysis 

100

The body experiences cellular starvation in DKA, leading to the breakdown of fats for energy; therefore, strategic administration of glucose is a critical part of treatment to address this underlying concept.

Nutrition

200

A nurse should anticipate administering this initially to correct severe dehydration in DKA.

Intravenous (IV) fluids

200

This age group is more susceptible to HHS, often triggered by an underlying infection.

Older Adult

200
Tachycardia, hypotension, dry mucous membranes, and poor skin turgor are all signs of this main clinical feature of DKA and HHS. 

Dehydration

200

This laboratory test directly measures the degree of acidosis.

ABGs

200

The extremely high glucose levels cause significant osmotic diuresis (glucose pulling water out of the body through the kidneys), causing alterations in this nursing concept.

Fluid and Electrolyte Imbalance
300

Besides high blood glucose, these acidic byproducts of fat metabolism are elevated in DKA.

Ketones

300

Patients with HHS typically have higher levels of this due to some residual insulin production.

Blood Glucose

300

This condition is triggered by a missed or insufficient dose of insulin, illness or infection, or can even be the first sign of undiagnosed Type 1 diabetes.

Diabetic Ketoacidosis

300

This laboratory test will measure the severity of electrolyte imbalances in patients with diabetic complications. 

Comprehensive metabolic profile (CMP)

300
A diagnostic indicator of DKA is acidosis, causing alterations in this nursing concept.

Acid-base imbalance

400

This rapid, deep breathing pattern is a compensatory mechanism for acidosis seen in DKA.

Kussmaul Respirations

400

Priority intervention for correcting an unstable blood sugar in HHS. 

Intravenous (IV) regular insulin 

400

This measures the amount of particles in the bloodstream versus the amount of fluid in the blood.

Serum Osmolality

400

This lab indicates an accumulation of unmeasured acids (like ketones) and is a critical diagnostic and monitoring tool for DKA severity.

Anion Gap

400

The compensating mechanism to help reduce the acidosis in the body by releasing excess CO2 impacts this related nursing concept.

Oxygenation

500

This imbalance is a hallmark of DKA, caused by the accumulation of acids.

Metabolic Acidosis


500

This should be administered when blood sugar stabilizes to less than 250 mg/dL. 

D5 1/2 NS
500

Both DKA and HHS patients can present with these "3 P”s of hyperglycemia.

Polyuria, Polydipsia, Polyphagia

500

Close monitoring and replacement of this electrolyte is crucial during HHS treatment to prevent life-threatening cardiac arrhythmias as insulin is administered.

Potassium

500

Long-standing diabetes, which causes neuropathy, also causes damage to small blood vessels (microvasculature), impacting this related nursing concept

Perfusion

M
e
n
u