What is hypoxemia? What are some s/s of it?
Hypoxemia is Low 02 level in blood circulation.
What is stable vs unstable Angina?
What are some priority outcomes for pancreatitis?
What can you educate on how to prevent immobility in PT?
What are some TX options for homonymous hemianopsia in acute setting?
What is a Pneumothorax?? What are some S/S of it?
Pneumothorax (PNThrx) is an accumulation of air in the pleural cavity that results in complete or partial collapse of the lung. Occurs in 50% of people with chest injuries. 2 types: Open (wound that allows air in and out freely when breathing) and Tension (Air enters the pleural space but does not escape causing pressure in the chest).
Aortic Stenosis: When the valve cusps of Aorta becomes fibrotic and calcify.
What nursing assessment points are priority post abdominal aneurysm?
Why would insulin be given during a stroke?
Due to PT's developing hyperglycemia in the acute phase of stroke may represent the body's stress in response to stroke; even w/o DM.
Explain the difference between arterial (PAD) vs circulatory(Venous) issue r/t toes??
What are the events of an asthma attack? At what point is respiratory failure imminent?
The sequence is as follows: Airway inflammation that is triggered by allergens or irritants> bronchoconstriction and edema, thickening of bronchial walls and the production of thick mucus> narrowed airways
!!! Respiratory distress is imminent when the is an Absence of Breath sounds!!
What are the primary Tx for essential and secondary HTN?
Primary HTN: 90-95% of all; Unknown cause but has many contributing factors: high sodium intake, DM, High BMI, and excessive ETOH.
S/S: Many people have no Sx. Occipital HA, light headedness, and epistaxis. Can also impair blood vessels in heart, kidney Dz, and blindness.
Tx: Goal: To gradually reduce PVR and BP (>140/90 mmhg)
What are some interventions for Liver cirrhosis PT?
Speed shock, damage to the veins/arteries, cardiac arrest, headache, tightness in chest, syncope
Why are COPD patient at risk for nutritional deficits?
What is CF? What are some s/s, Tx options, and nursing care?
Cystic fibrous: hereditary disorder, dysfunction of the exocrine glands and the production of thick, tenacious mucus
S/S: cough= 1st sign, productive thick and purulent, obstructs airway, Obstruction of pancreatic ducts: unable to provide pancreatic enzymes to GI track= Pts develop diabetes, Unable to absorb proteins, fats, and fat-soluble vitamins, Stools= bulky and foul smelling
Tx: pancreatic enzyme replacement, CPT, and aerosol and neb txs to reduce mucus viscosity
Nursing care/ goals: effective airway clearance, prevention or tx of infection, adequate nutrition, and effective self-health management by pt and fam
Describe how a drop of blood travels through body? Start from the right atrium.
Deoxygenated blood flows from the right atrium, through the tricuspid valve, to the right ventricle. From the right ventricle the blood flows through the pulmonary valve, to the pulmonary artery, leading to the lungs where the blood picks up oxygen. Oxygenated blood flows back to the heart from the lungs through the pulmonary veins, dumping into the left atrium. From the left atrium, blood travels through the mitral valve (bicuspid), to the left ventricle. From the left ventricle, blood flows through the aortic valve, through the aorta, carrying oxygenated blood to the rest of the body.Arteries>areterioles> Capillaries>Venioles> Veins> Deoxygenated blood comes back from the cells to the superior and inferior vena cava, dumping the blood back into the right atrium.
What is hepatic encephalopathy and what are some S/S and Tx?
What are some nursing actions for IV vesicant use??
Drugs that are especially toxic to subq tissues are called vesicants. Common drugs are vasopressors, potassium chloride, antineoplastic agents, and any solution that has a PH <5 or >9 i.e. D10W, calcium gluconate, Dobutrex, and amphotericin B (fungizone)
S/S: similar to that of infiltration, area may be pale and puffy, can feel hard and cool.
What are possible Nursing Dx for CVA? Note 3 interventions with rationale
inadequate oxygenation: elevate the head of bed 25-30 degrees to improve oxygenation, good hydration to thin respiratory secretions for easier expectoration of secretions that may be blocking the airway, suctioning and frequent position changes can help to prevent aspiration and promote removal of secretions.
Potential injury r/t seizure activity, confusion, motor & sensory impairment, increased ICP, hemorrhage after recombinant tissue plasminogen activator administration.
it is important to raise the head and side rails & pad them according to agency protocol to reduce trauma if the patient strikes the rails
safety precautions are essential for confused pts. orient pt surroundings and explain why they should not get up unassisted
a bed check system may be of more benefit when the patient is having difficulty understanding directions.
get sufficient help or use mechanical devices to assist the patient in and out of the bed.
The cornea is susceptible to injury when not protected & in healthy patients, kept moist by the closed eyelid & the blink reflex. artificial tears may be used to provide moisture.
no not place objects where the patient cannot see or feel & check those areas frequently.
monitor for s/s of ICP: change in LOC, elevated BP, deterioration of motor function, new HA, n/v.
medical interventions: diuretic & barbiturate drugs, hyperventilation, surgical intervention.
potential for fluid imbalance r/t inadequate inadequate intake or excessive diuresis
monitor labs: specific gravity, electrolytes & hematocrit.
look for s/s of fluid volume excess (s/s later in the guide) report to the RN and explain to the pt and family why the pt may need to be on the fluid restriction.
monitor for signs of fluid volume deficit (s/s later in the guide)
place fluids within sight of the pt on the non-paralyzed side. may need to use spill proof containers, feed slowly if pt has dysphasia.
have suction equipment setup, use thickening agents if necessary.
inadequate nutrition r/t dysphasia, inability to feed self, inability to chew.
pt may need assistance feeding.
if pt had a NG or peg you must check placement, be certain formula & rate is correct. keep the head slightly elevated to prevent aspiration. gently cleanse the nairs several times a day. if diarrhea occurs, notify PCP and formula may need to be changed.
pt may develop hyperglycemia so make sure s/s are monitored.
elevated body temp r/t effects of neurologic impairment and/or metabolic process.
monitor temp and tx elevations properly with antiemetic drugs. always assess for underlying infection.
potential for impaired cognition r/t inadequate cerebral circulation
be sure that eyeglasses and hearing aids work and are worn if the pt normally wears them.
place clocks and marked calendars in view.
instructions and information should be concise & repeated as needed.
a familiar person can be helpful with a confused pt. if the visitor is startled, provide guidance to the visitor on how to deal with the behavior.
inability to communicate effectively r/t aphasia
use questions that can be answered with “yes” or “no” may allow the pt to respond more easily. pause attentive and allow the pt to respond.
speech therapy may be initiated once the medical condition is stable.
there are about 15 ND and NI on page 411-416
What is COPD? What are some S/S, complications, Tx options, or pertinent Nursing care
COPD: umbrella term for chronic bronchitis, emphysema, irreversible refractory asthma. increased production of mucus and chronic cough that persist for at least 3 months of the year for 2 straight years. Mucous obstructs airways & is trapped.
Emphysema: degenerative, nonreversible disease, enlargement of the airways beyond the terminal bronchioles. 2 types. may have one or both types
Complications: respiratory failure and heart failure, Infection, air pollution, continued smoking, left ventricular failure, MI, PE
S/S: chronic bronchitis: color dusky to cyanotic, recurrent cough, hypoxia, acidosis, edematous, external dyspnea, hypercapnia (↑ RR), heavy cigarette smokers, digital clubbing.
Pulmonary emphysema: ↑ CO2 retention, pursed lip breathing, dyspnea, skinnier, barrel chest, ineffective cough, orthopneic, external dyspnea, speaks in short sentences, anxious, use accessory muscles, leads to right sides HF, can only situp.
Med dx: H&P, reports of clinical manifestations, physical exam, PE, PFT (most reliable), MRI, CT, ABGs, spirometry w/bronchodilator, 6 min walk test w/ pulse ox.
Med tx: GOAL= maintain symptom relief, slow disease progress, improve exercise tolerance, prevent /tx complications, START: stop smoking, drug & O2 therapy, pulmonary rehab & surgery.
Tx for respiratory failure: Hospitalized at least yearly for exacerbations
s/s:changes in dyspnea, cough, sputum; r/t: smoking, infection, air irritants, heart failure, not taking meds
Tx: noninvasive positive pressure ventilation (NPPV), chest PT, aerosol bronchodilators, O2 therapy
Surgical: Bullectomy, Lung transplant
LVRS: lung volume reduction surgery, removal of 30% of the hyperinflated lung tissue so that remaining lung tissue can function better
Nursing care: obtain med hx, physical exam, eval meds, note posture/ skin color/ resp effort/ accessory muscles, assess ADLs, VS, pure lip breathing, chet shape, nail beds.
Interventions: VS, ABGs, tachycardia, tachypnea, ↑CO2, ↓pH, increase meds, edu on smoking, no more than 2-3 L/ min O2, pursed lip breathing, admin bronchodilators.
Describe what EKG will show with these changes:
1. Rate slower than 60 BPM, all other characteristics WNL?
2. Rate Greater that 100 BPM, all other characteristics WNL, PT feels palpitations?
3. No Rate, no characteristics, QRS/T wave not present
4. Rate between 100-250 BPM, Regular P wave/PR interval are absent, QRS is wide, appears bizarre, may appear like PVCs but T Wave opposite deflection to QRS.
5. Atrial Rate greater than 400 if it can be determined, Ventricular rate 100-150, can appear regular or irregular, No P waves or PR, QRS normal, T wave undeterminable. Baseline between QRS complex is wavy
6. Rate usually normal; irregular due to PVCS, No P wave with PVC, QRS wide and bizarre, T wave opposite deflection to the PVC
1. Sinus Bradaycardic
2. Sinus Tachycardia
3. Asystole
4. Ventricular Tachycardia
5. Atrial fibrillation
6. Premature Ventricular Contractions PVCs
What is the difference between a PEG tube and TPN? What specific procedures should you follow when administering? What are some S/S of complications with them?
The key difference between TPN and tube feeding is that total parenteral nutrition or TPN refers to the supply of all daily nutrition directly into the bloodstream, while tube feeding refers to the supply of nutrition through a tube that goes directly to the stomach or small intestine.
PEG Tube:
Complications: diarrhea, nausea ← stop the tube feeding if this occurs, abdominal distention, hyper/hypoglycemia, constipation, dehydration, skin issues around the tube, peritonitis, obstruction or displacement of tubing, fluid imbalance
TPN:Intravenous feedings can only provide: water, glucose, electrolytes, minerals & vitamins. Do not give any meds through. Why not peripheral: The superior vena cava rapidly diluted the hypotonic solution by the large, fast flowing volume of the blood it pumps. Meanwhile, never given through smaller veins because it would cause thrombophlebitis (inflammation of the vein) & not enough volume to dilute the solution. (PG 246)
Complications: s/s of blood glucose changes: diaphoresis, lethargy; hyperglycemia (s/s sweet taste in mouth, confusion, polyuria) causes the pancreas to secrete more insulin may then cause hypoglycemia.
infection at the site: redness, swelling, foul odor or purulent drainage, fever
circulatory overload: blood glucose or excessive diuresis
pulmonary complication, injury to the veins & arteries surrounding the TPN cath site, air embolism, infection, electrolyte imbalance, mineral deficiency.
never use a TPN line to administer drugs
DO NOT SPEED UP THE RATE TO CATCH UP IF YOU FALL BEHIND
You have a multitude of PTs and various ABG results, Decipher which? Double points for compensation?
PT 1: PH:7.60, PaCO2: 40, HCO3: 33
PT 2: PH:7.27, PaCO2: 50, HCO3: 26
PT 3: PH:7.35, PaC02: 27, HCO3: 18
PT 4: PH:7.58, PaCO2: 32, HCO3: 15
PT 1: Metabolic Alkalosis, no compensation
PT 2: Respiratory Acidosis, no compensation
PT 3: Metabolic Acidosis with Compensation
PT 4: Respiratory Alkalosis with compensation
What are some important PT teaching topics for HCTZ, Albuterol, Cardizem, and Lasix?