Shock Me Maybe
This cardiac chamber sends deoxygenated blood to the lungs and is most affected by increased pulmonary resistance.
Right ventricle ( Cardiology Slide 8)
This medication is given 324 mg PO (typically four 81-mg chewables) for suspected acute coronary syndrome.
Aspirin ( Drug Cards)
This hormone is released by alpha cells of the pancreas and raises blood glucose by converting stored glycogen back into glucose.
Glucagon ( endocrine slide 20 and 27)
This route of medication administration is enteral, involves the GI tract, and is affected by first-pass metabolism, making onset slower than IV routes.
Oral (enteral) administration (toxicology slide 3-6)
End-tidal CO₂ represents the exhaled “exhaust” of metabolism and depends on oxygenation, perfusion, and ventilation.
Carbon dioxide (CO₂) ( Respiratory emergencies slides 2-4)
According to the Starling Law, increasing preload will have what effect on stroke volume?
It increases stroke volume by increasing the force of contraction (Cardiology slides 9-10)
This commonly used analgesic is contraindicated in liver failure and becomes toxic at single doses over 150 mg/kg.
Acetaminophen (Drug Card)
These three classic findings—polyphagia, polydipsia, and polyuria—are commonly associated with this metabolic condition.
Hyperglycemia ( endocrine slide 41)
This pharmacologic concept describes a drug that binds to a receptor and activates it, producing a physiologic response.
Agonist ( toxicology slides 20-21)
This device provides qualitative confirmation of CO₂ presence, turning yellow when CO₂ is detected but does not measure an actual value.
Colorimetric CO₂ detector ( respiratory emergencies slides 5-6)
During diastole, blood flow to which structures is critical for myocardial oxygenation?
Coronary arteries ( cardiology slide 18)
This bronchodilator may cause tachycardia, tremors, and anxiety, and should be monitored using EtCO₂ and lung sounds during administration
Albuterol ( Drug Card)
This diabetic emergency presents with rapid, deep respirations, fruity breath odor, and is caused by fat metabolism producing ketones.
Diabetic ketoacidosis (DKA) ( endocrine slide 42)
These chemical messengers are responsible for pain, fever, and inflammation, and are inhibited by NSAIDs.
Prostaglandins ( toxicology slides 11-12)
The normal EtCO₂ range for most patients is 35–45 mm Hg, while patients with COPD may have a higher baseline due to CO₂ trapping.
Normal EtCO₂ range ( respiratory emergencies slide 8)
This type of angina is caused by coronary artery spasm, often occurs at night, and has minimal risk of myocardial infarction.
Prinzmetal (variant) angina ( Cardiology slides 58,60)
This medication used for chest pain is contraindicated if systolic blood pressure is under 100 mmHg or if the patient has used ED drugs within the last 24–48 hours
Nitroglycerin (Drug Card)
Unlike DKA, this hyperglycemic emergency typically develops over days to weeks, occurs most often in Type 2 diabetes, and involves profound dehydration without ketone production.
Hyperglycemic hyperosmolar state (HHS) ( endocrine slide 43)
A patient with hot, flushed, dry skin, mydriasis, tachycardia, and agitation is most consistent with this toxidrome.
Anticholinergic toxidrome ( toxicology slides 47-50)
A sudden drop in EtCO₂ after airway placement should prompt evaluation using this mnemonic: Dislodged tube, Obstructed tube, Pneumothorax, Equipment failure.
DOPE ( respiratory emergencies slide 23)
In left-sided heart failure, blood backs up into the lungs causing which classic assessment finding?
Pulmonary edema with pink, frothy sputum (Cardiology slide 90)
This opioid antagonist may cause acute agitation, vomiting, and rarely dysrhythmias or pulmonary edema after administration.
Naloxone (Drug Card)
This life-threatening endocrine emergency is caused by too little cortisol and aldosterone and can result in hypoglycemia, hypotension, and hyperkalemia.
Addisonian crisis ( endocrine slide 52)
This type of hypoxia occurs when oxygen cannot be used at the cellular level, even though oxygen levels and skin color may appear normal, as seen in cyanide poisoning.
Histotoxic hypoxia ( toxicology slides 131-134)
During cardiac arrest, an EtCO₂ reading greater than 20 mm Hg indicates effective chest compressions, while a sudden rise of 8 mm Hg or more suggests this event.
Return of spontaneous circulation (ROSC) ( respiratory emergencies slide 45)