Anatomy
Which SCI
Shock Me
It's Complicated
(Blank)
100

cervical vertebrae

7

100

a patient with no demonstrable sensory or motor function below a certain level

complete SCI

100

This can occur in injuries to T6 and above

Neurogenic shock

100

signs and symptoms of autonomic dysreflexia

severe headache, bradycardia, facial flushing, pallor and cold skin, diaphoresis of the lower part of the body

100

This affects approximately 90% of patients with spinal cord injuries

Neurogenic bladder

200

thoracic vertebrae

12

200

incomplete SCI

some degree of motor or sensory function remains

200
Neurogenic shock

loss of vasomotor tone and sympathetic innervation to the heart

200

autonomic dysreflexia causes

stimulus originating below the level of injury

200

patients with c spine or upper t spinal cord injures are at a high risk of this

potential respiratory failure (diaphragm vs intercostal muscle paralysis)

300

lumbar vertebrae

5

300

complete motor loss of function below the level of injury; loss of pain and temperature sensation below the level of injury

anterior cord syndrome

300

your patient is unable to move any extremities, however you note spasticity occurring. what is this patient experiencing 

spinal shock

300

inflammation of tissues which can lead to permanent loss of function without proper intervention

secondary SCI

300

decreased GI motility can lead to these

SBO, ileus

400

2 most vulnerable regions of the spine

cervical spine , thoracolumbar junction

400

central cord syndrome

loss of motor and sensory function in the upper extremities that is greater than that of the lower extremities

400

what type of shock is neurogenic shock

distributive

400

vasogenic edema

fluid buildup

400

this assessment can assist in rapidly identifying an expanding hematoma/increased swelling

neuromuscular assessment

500

area of skin innervated by the sensory axons within a particular segmental nerve root

dermatome

500

partial transection of the spinal cord causes this syndrome

brown-sequard syndrome

500

how do we manage neurogenic shock?

pharmacological interventions (pressors, midodrine, maintenance of normovolemia

500

cellular metabolism disruption resulting in swelling

cytotoxic edema

500

decreased sensation, inability to change position can lead to this

pressure injury development

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