This contraindication for ice also means a loss of sensation
Anesthetized skin
This is the term for a loss of a painful sensation
Analgesia
This is the first step in the inflammation process
Hemostasis (vasoconstriction & clotting)
This is the effect superficial heat has on fluid
Decreased viscosity
This is the first stage of the Grief Model
Denial
This is the impact ice has on cellular metabolism.
These nerves transmit "slow" pain
C-Fibers
This is the time frame the remodeling phase takes
Up to 1 year
This is how deep superficial heat can reach
Less than 2 cm
This is the temperature range for cold modalities in Celsius
15 - 18 degrees C
These pain fibers are myelinated
A-Delta Fibers
These cells are begin building collagen after an injury
Fibroblasts
This is the therapeutic range for heat modalities
104 - 113 degrees Fahrenheit
This is the psychological intervention that is well supported in the literature to improve motivation
Goal setting
The risk of frostbite is a major concern for this type of cold modality
Chemical cold pack
This is the neurotransmitter that "closes the gate"
Enkephalin
This is the scientific term for why swelling occurs after injury (think simple)
Osmosis (specifically interstitially)
This condition is an indication that the treatment was too hot
Mottling
This is the main difference between compliance and adherence
Voluntary on the patient's side of things
This phenomenon is the theoretical purpose behind applying ice after an acute injury
Secondary cell death
Things like gum chewing and oscillating joint mobilizations help control pain via this theory
Rhythmical Pain Modulation
This is the chemical mediator that causes vasodilation and is the hallmark of inflammation
Histamine
This is how often you should clean a whirlpool as long as all patients do not have any open wounds
Once a day
These are the three components of Nagi's model of injury and response