the basics
sources of burns
classifications of depth
Assessments
PHASES
100

who is at risk for burns?

highest risk for death?

the very young and very old

elderly (>60), >40% TBSA burn, inhalation injury


100

THERMAL

2 TYPES- WHAT ARE THE TYPES? GIVE EXAMPLES

DRY- open flame, explosions

MOIST- liquid, steam

100

1ST DEGREE : Superficial Thickness

Damage only to?

Caused by exposure to ...?

What does it look like??

WHEN DOES IT HEAL?

damage only to EPIDERMIS 

caused by exposure to high heat for short duration and low heat for long duration

PINK TO RED, NO EDEMA/PAIN, NO BLISTERS

Skin peels and heals in 1 week

100

Cardiovascular assessment

body thinks its in...?

occurs in 1st (?) and can continue to ?-? hours

hypovolemic shock

1st 12 hrs

can cont to 24-36 hrs

100

1st phase- emergent/resus

what 2 labs do we look at in first 24-48 hrs?

what can we give in first 24?

WHEN DO WE INTUBATE?

INCREASED H+H, HYPONATREMIA, HYPERNATREMIA

1st 24 albumin or synthetic plasma expanders

INTUBATE IF AIRWAY SWELLING IS SUSPECTED


200

#1 priority for burns

closure of wound and prevent infection

200

CONTACT

Direct contact w/...? 

hot metal, tat, grease 

200

2ND DEGREE: SUPERFICIAL AND EXPOSED/DEEP

Involves entire ..? and parts of ...?

Caused by?

SUPERFICIAL- what does it look like? when does it heal?

EXPOSED/DEEP- what does it look like? what might it need? when does it heal?

caused by scalding, flames, brief contact w hot objects

superficial - pink-red, moist w/ edema. heals in 2 weeks

exposed- red and moist, blanching and blistering, some pain, eschar soft and dry. MAY NEED GRAFT, heals 2-6 weeks

200

cardiovasc assessment

what will we see?

TACHY

ELECTROLYTE IMBALANCE

HYPOTENSION

FLUID OVERLOAD

HYPERKALEMIA

HYPONATREMIA

INCREASED H AND H

200

1ST PHASE CONTINUED

what med to prevent infection?

analgesics? what to monitor?

anesthetics?


tetanus

opioids- monitor for resp dep

ketamine

300

burns cause difficulty....?

which leads to an increase in (?) to maintain (?)

difficulty regulating body temp, inability to sweat, and changes in sensory function

increase in metabolism to maintain body heat 

300

CHEMICAL

Exposure to what kind of agents?

Severity depends on?

Flush w...?

COVER BURNED AREAS W...?

exposure to cleaners, bleach, drain cleaner and agents in an industrial setting

severity depends on duration of contact, concentration of chemical, amount of tissue exposed, and chemicals action

flush with large amounts of water

cover burned areas with DRY STERILE DRESSING

300

3RD DEGREE- DESTROYED INTO ...?

what does it look like?

requires...?

how long to heal?

DESTROYED INTO FAT

any color, severe edema, no blistering/pain, eschar hard and inelastic

REQUIRES GRAFTING

weeks to months for healing

300

What do we do for a cardiovascular assessment? USING WHAT?

Monitor..?

WHAT MIGHT THEY NEED?

FLUID RESUS W 2 LARGE BORE IV 

daily weights, urine output (0.5 ml/kg/hr), monitor for s/s of fluid overload

may need albumin and blood products

300

1ST PHASE THERMOREGULATION FOR DECREASED TEMP

NURSING CARE?

Low grade fever in first few hours can stay increased for how long?

WARM INSPIRED AIR

WARM ROOM AND BLANKETS

FLUID WARMER

KEEP WOUNDS COVERED AND WORK QUICK


increased for several weeks

400
Burns can occur on ...?

orofacial burns cause ....?

burns can occur on inhalation

orofacial burns cause edema that impairs breathing


400

ELECTRICAL

what is the iceberg effect?

The electrical current penetrates skin and...?

What labs do we monitor?

Hook them up to (?) and watch for (?)

iceberg- surface injuries look small, internal injuries LARGE

penetrates skin, flows thru body and causes damage until it exits the body

MONITOR CARDIAC ENZYMES AND ABGS

Hook up to EKG watch for arrythmias

400

4TH DEGREE- MUSCLE AND BONE

what does it look like?

Graft does not...?

Healing?

Black, severe edema, no blistering and no pain, eschar present 

GRAFT DOES NOT WORK

TAKES WEEKS TO MONTHS FOR HEALING OR NEVER!

400

RESPIRATORY ASSESSMENT

when do we suspect a respiratory burn? what will we see?

singed nasal hair/eyebrows

drooling

wheezing

edema of nasal septum

smokey breath

hoarseness

sooty septum

400

1ST PHASE 

whats decreased?

whats increased?

myoglobin release=?

THESE PATIENTS NEED..?

decreased- sodium, albumin, wbc

increased - edema, K, and HCT

myoglobin release= aki and hypovolemic shock

THESE PTS NEED MASSIVE FLUID RESUS

500
ITS CRITICAL TO ASSESS ....?


LISTEN FOR....?

assess mouth nose and throat

listen for cough, sob, and hoarseness


500

RADIATION

caused by?


sunburns, cancer tx, working in nuclear industry

500

METABOLIC AND IMMUNOLOGIC

? occurs due to stress response

? cal/day .... DOUBLE TRIPLE ?-? days after burn

increase what?

HYPERMETABOLISM

5000 cal/day. DOUBLE TRIPLE 4-12 days after injury

INCREASE PROTEIN AND CARBS

500
CARBON DIOXIDE POISONING

what is a risk?

>?% indicates smoke inhalation

Manifestations? TX?

INHALATION INJURY

>10%

HALLMARK- CHERRY RED LIPS AND CHEEKS

sloughing mucosa in resp tract, nausea, tinnitus, upper airway edema

TX- Lab draw COHB lvl, high flow 02 for 6 hr

500

2ND PHASE- ACUTE PHASE

begins when? until when?

H+H?

NA?

K?

WBC?

GLUCOSE?

ABG?

TOTAL PROTEIN/ALBUMIN?

Begins 36-48 hrs AFTER INJURY

h+h- decreased

na- decreased

k- decreased

WBC- inital increase then decrease 

glucose- increased

ABG- slightly hypoxic and metabolic acidosis

Pro/alb- decreased