This system is responsible for “fight or flight” responses such as increased heart rate and bronchodilation.
What is the sympathetic nervous system?
(ANS sympathetic activation increases HR, dilates bronchi, and decreases GI activity.)
These drugs mimic sympathetic nervous system effects.
What are adrenergic agonists (sympathomimetics)?
These two vital signs must be closely monitored when giving adrenergic agonists.
What are heart rate and blood pressure?
This system promotes rest, digestion, and energy storage.
What is the parasympathetic nervous system?
(Parasympathetic activity slows HR and increases GI motility and secretions.)
This medication is commonly used in cardiac arrest and anaphylaxis.
What is epinephrine?
This is a major cardiovascular risk when giving adrenergic medications.
What are dysrhythmias or tachycardia?
These receptors primarily increase heart rate and contractility when stimulated.
What are beta-1 receptors?
This medication can cause CNS sedation and dry mouth because it reduces sympathetic outflow
What is clonidine?
These drugs should be used cautiously in patients with hypertension or heart disease.
What are adrenergic agonists?
These receptors cause bronchodilation and uterine relaxation when stimulated.
What are beta-2 receptors?
This medication is used to stop premature labor by relaxing uterine smooth muscle.
What is a beta-2 agonist (example: terbutaline)?
This route requires careful site monitoring due to risk of tissue necrosis with some vasoactive drugs.
What is the IV route?
This neurotransmitter is released by most sympathetic post-ganglionic neurons.
What is norepinephrine?
This is the priority treatment if IV epinephrine extravasates into surrounding tissue.
What is phentolamine administration?
This is why clonidine should not be stopped abruptly.
This is why clonidine should not be stopped abruptly.
This drug class works by blocking sympathetic receptors and lowering blood pressure and heart workload.
What are adrenergic blockers (sympatholytics)