ECG Interpretation
ECG Quick Facts
ECG Interpretation
ECG Quick Facts
STEMI Location
100

Sinus rhythm ~75bpm with Wolff Parkinson White morphology

100

Describe the electrical conduction pathway of the heart. 

Beginning at the sinoatrial node (SA node), conducting through the atria to the atrioventricular node (AV node) through the bundle of His, down the left and right bundle branches and to the purkinjie fibres.

100

Ventricular standstill

100

What are the rate parameters for sinus bradycardia, sinus rhythm and sinus tachycardia? 

Bradycardia <60bpm
NSR 60-100bpm
Sinus tachycardia >100bpm

100


Extensive Anterior STEMI 

"Tombstone" morphology STE throughout precordial leads and high lateral leads. 

200

Sinus rhythm with CHB and  ventricular escape rhythm at 40bpm. 

200

Describe 2nd Degree AV block. 

Hint: There is 2

Wenckebach/Mobitz I: progressive lengthening of the PR interval until a non-conducted p-wave occurs.

Mobitz II: intermittent non-conducted P waves WITHOUT progressive prolongation of the PR interval

200

Sinus rhythm 75bpm with LBBB

200

Describe the mechanism of the R on T phenomenon 

A PVC occurs during the vulnerable relative refractory period of the preceding cardiac cycle, inducing VF
200

STE in inferior leads, reciprocal changes anterior leads

Inferior infarct 

300

Junctional rhythm 38bpm

300

When and Who reported the first ECG?

1903 by Willem Einthoven 

300


Atrial fibrillation in a patient with WPW (pre-excitation). 

Rates up to 300bpm. 

300

Explain the clinical significance of a patient with pre-excited atrial fibrillation

The presence of an accessory pathway allows for rapid conduction to the ventricles, bypassing the AV node - rapid ventricular rates may result in degeneration to VT or VF. 

300


Anterolateral STEMI

STE anterior leads (V2-V4) and lateral leads (1, aVL, V5-6)

Reciprocal STD in inferior leads. 

400

PVC on T wave (R on T) causing Torsades de Pointes 

400

Demonstrate and describe the placement of a 12 Lead ECG using correct anatomic terms. 

400

Sinus rhythm with AV dissociation.

400

What mechanisms may cause a wide complex during an SVT? 

Pre-excitation (accessory pathway) 

Rate related bundle branch block 


400


Inferior STEMI

Hyper-acute T waves and STE in II, II and aVF

Reciprocal changes in anterior leads. 

500

Hint: 18 Y/O presenting with 1/7 chest pain

Sinus rhythm with global STE, STD in V1 

Myopericarditis 

500

How long is a prolonged QT interval and what arrhythmia are people with a long QT at risk of developing?

Prolonged if >440ms in men and >460ms in women. Torsades de pointes.

500

Atrial fibrillation with RBBB, 92bpm 

500

Describe the regions of an ECG in relation to localising acute coronary syndrome. 

500


Inferolateral STEMI

STE in inferior (II, III and aVF) and lateral (I, V5-6)

STD in V1-V3 suggesting posterior extension. 

600

Artifact

600

What ECG findings would you see associated with sinus node dysfunction? 

Hint: there is 8 

Sinus bradycardia
Sinus arrhythmia
Sinus pause or arrest
Sino-atrial exit block
Chronotropic incompetence
Tachy/brady syndrome
Wandering Atrial Pacemaker
Atrial fibrillation

600

Monomorphic VT, shock on T wave causing VF, shock reverting to sinus rhythm. 

600

What is the lead on the top right of Einthoven's triangle? 

Right arm 

600


Anterior STEMI 

STE V1-6 

Reciprocal changes III and aVF