Fluid Balance Pt I
Electrolytes—Calcium
Electrolytes—Sodium
Electrolytes--K+ and Mg ++
Fluid Balance Pt II
100

What interventions would you implement for a patient with fluid volume excess?

Monitor daily weight (weight loss indicates successful intervention)

Administer diuretics and O2 as ordered

Fluid and sodium (Na) restrictions

Monitor I & O and put in Fowler's position


100

What is the normal range for serum calcium?


9.0–10.5 mg/dL

100

What is the normal range for sodium (Na+)?

135 to 145 mEq/L

100

What is the normal range for potassium?

What is the normal range for magnesium?

3.5 to 5.0 mEq/L

1.3 to 2.1 mEq/L

100

The main intracellular cation?

Potassium (K+)


200

What interventions would you implement for a patient with deficient fluid volume?

Monitor daily weight (most accurate way to evaluate fluid balance)

Monitor I & O

Increase fluid intake (PO and IV if severe)

Treat underlying cause


200

What are the signs and symptoms of hypocalcemia?

Neuromuscular excitability: Spasms of face, lips, larynx (can cause stridor); Tingling sensation around the mouth; Mental status changes; Increased and irregular heart rate which can lead to cardiac arrest; Hypotension; Trousseau's sign, Chvostek's sign.

200

Who is at risk for developing hyponatremia?

Patients with NPO status, use of diuretics, GI suctioning (especially continuous), decreased aldosterone, excess ingestion of hypotonic fluids.

200

What are the signs/symptoms of hypomagnesemia?

What population is especially vulnerable to this?


S/S are similar to low Ca+ and K+ levels.  Tetany, Chvostek's sign, Trousseau's sign, tremors, muscle spasms, and cardiac arrhythmias.

Alcoholics are especially vulnerable. 

200

The main extracellular cation?

Sodium (Na+)

300

What signs/symptoms would you expect to see in a patient with fluid volume excess?


Bounding pulse, increased BP, weight gain, increased and dilute urine output, increased and shallow respirations.  May see neck vein distention, pitting edema of lower extremities, crackles in the lungs.

300

Why is it important to monitor calcium levels following a thyroidectomy?

Parathyroid glands can be accidentally removed during a thyroidectomy causing hypocalcemia.

The parathyroid gland secretes a parathyroid hormone that regulates calcium levels in the blood by increasing the levels when they are too low. 

Staff should always have calcium gluconate available post-operatively.


300

What are the symptoms of hyponatremia?


It can be vague and depends on whether it has a fluid imbalance.  Severe hyponatremia presents with weakness, N/V/D, and mental status changes such as confusion, disorientation, and personality changes due to low sodium and osmolarity, which causes more water to collect around the brain.

300

What are the signs/symptoms of hypokalemia?


S/S: Anorexia, cardiac arrhythmias, leg cramps, muscle fatigue, weakness, postural hypotension

ECG: prolonged PR interval, flattened T wave, and prominent U wave

300

What factors can lead to edema?

Increased blood volume = increased hydrostatic pressure

Lower albumin = reduced osmotic pressure

400

What is included in a hydration assessment of a patient?

Monitor daily weights.

Assess for skin tenting (forehead or sternum)

Assess mucous membranes and tongue for moisture and furrows

Assess for thirst and urine output


400

What are the signs and symptoms of hypercalcemia?


Increased BP and pulse, skeletal muscle weakness, decreased GI motility including diarrhea, severe fatigue, confusion.

400

What are the signs/symptoms of hypernatremia?

Thirst, mental status changes due to too little water in and around the brain, tremors, seizures, muscle weakness

400

What are the signs/symptoms of hyperkalemia?

Patient presentation depends on K+ elevation and if chronic or acute elevation.

Early symptoms: numbness, muscle cramps, diarrhea; ECG: wide QRS, tall and peaked T waves, bradycardia; Can lead to cardiac arrest.

400

What are the steps in the Renin-Angiotensin-Aldosterone System?

What initiates this system?


1. Renin released from kidneys: Converts angiotensinogen (from the liver) to angiotensin I

2. Angiotensin I converted to angiotensin II: In lungs by angiotensin-converting enzyme (ACE)

3. Angiotensin II (vasoconstrictor): Activates adrenal cortex to release aldosterone

4. Aldosterone: Increases sodium and water reabsorption and potassium secretion by kidneys

Hypotension, hypovolemia, low cardiac output.

500

What labs and signs/symptoms would you expect to see in a patient with dehydration?

Increased Hct and BUN, increased urine specific gravity, low BP, rapid pulse, increased temperature, decreased urine output, skin tenting, dry skin, and mucous membranes.

500

What are the causes for hypercalcemia?

•Elevated PTH

•Cancer (malignant cells causing bone destruction)

500

What are the interventions for hypernatremia?

Monitor weight, monitor I & O, administer diuretics, restrict dietary sodium 

500

What are food sources of potassium?

Potatoes, sweet potatoes, soybeans, bananas, tomato juice, dried apricots, kidney beans, orange juice, and spinach.

500

This is the most abundant glucocorticoid. In large doses, it has both glucocorticoid (glucose-elevating and antiinflammatory) and mineralocorticoid (sodium-retention) effects.

What is cortisol?