AXON
FIBER THAT EMERGES FROM CELL BODY AND PROJECTS TO TARGET CELLS
CHANNELS
TRANSMEMBRANE PROTEINS THAT MAKE TRANSPORTATION POSSIBLE
MEMBRANE POTENTIAL
ELECTRIAL STATE OF THE CELL MEMBRANE THAT CAN HAVE SEVERAL VARIATIONS
DEPOLARIZATION
MEMBRANE MOVE TOWARDS ZERO
NEUROTRANSMITTERS
CHEMICAL MESSENGER THAT CONVEYS UPSTREAM INFORMATION TO DOWNSTREAM TARGET CELLS
PRESYNAPTIC CELL
AXON TERMINAL 'BEFORE' SYNAPSE
ELECTROCHEMICAL GRADIENT
POTENTIAL STORED ENERGY THAT CAN BE PUT TO WORK TO MAKE GREAT THINGS HAPPEN
IF THE CELL IS AT REST, IS THE MEMBRANE OPEN?
NO, THE MEMBRANE IS CLOSED
INACTIVATION GATE
COLLECTION OF AMINO ACIDS THAT PREVENT DEPOLARIZATION
ACETYLCHOLINE
MAIN NEUROTRANSMITTER AT NEUROMUSCULAR JUNCTION, PARASYMPATHETIC NERVOUS SYSTEM AND FRONTAL LOBE
POSTSYNAPTIC CELL
'DOWNSTREAM' MEMBRANE
LIGAND-GATED CHANNELS
OPENS BECAUSE SIGNALING MOLECULE LIGAND BINDS TO EXTRACELLULAR REGION OF THE CHANNEL
CYTOSOL
HIGH CONCENTRATIONS OF ANIONS, FORM OF PHOSPHATE IONS AND NEGATIVE CHARGED PROTEINS
REPOLARIZATION
MEMBRANE VOLTAGE MOVES BACK TOWARDS -70mV, VALUE OF RESTING MEMBRANE POTENTIAL
IONOTROPIC RECEPTORS
'BUILT IN' ION CHANEL THAT WILL OPEN/CLOSE WHEN NEUROTRANSMITTER BINDS
NISSL BODIES
WHERE PROTEINS ARE MODIFIED AND SENT TO OTHER ORGANELLES AT THE END OF MEMBRANE SYSTEM
VOLTAGE GATED CHANNEL
RESPONDS TO CHANGES IN ELECTRICAL PROPERTIES OF MEMBRANE (EMBEDDED)
WHAT IS THE CHARGE OF RESTING MEMBRANE POTENTIAL
-70mV
WHAT HAPPENS TO SODIUM WHEN CHANNELS INACTIVATE?
SODIUM INFLUX STOPS
METABOTROPIC RECEPTORS
ACT ON ION CHANNELS THAT AREN'T BUILT IN
NUEROFIBRILS
REINFORCEMENT OF NEURON REGIONS AND MAINTAIN SPECIALIZED ANATOMY
PLAYS A ROLE IN AXON DIAMETER REGULATION
NON-GATED CHANNEL
RANDOMLY GATED (OPEN/CLOSES RANDOMLY)
POTASSIUM'S HAPPY NUMBER
-90mV
T OR F: ABSOLUTE REFRACTORY PERIOD IS ASSOCIATED WITH HYPERPOLARIZATION PHASE?
F: ABSOLUTE REFRACTORY PERIOD IS ASSOCIATED WITH DEPOLARIZATION AND REPOLARIZATION
WHAT ARE THE TWO NEURONAL SYNAPSES
EXCITATORY POST-SYNAPTIC POTENTIAL
INHIBITORY POST-SYNAPTIC POTENTIAL