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Defensive Driving
Got You Covered
Weird Stuff
Bite Marks
Nothing to stand on
100
Attachment loss, very rapid with massive bone loss, can be early onset, localized or generalized, rapidly progressing or refractory...look at other family members
What is Aggressive P-itis
100
Bone loss usually 30%, rapid, AA, Prevotella Intermedia
What is Refractory P-itis
100
Non-AIDS type/Patient with nutritional deficiencies or Dental Hygiene Students in their 3rd semester!
What is NUG
100
Position of the mandible where there is maximum interdigitation of the teeth (Home Base for your teeth)
What is Centric Occlusion
100
Loss of up to 1/3 of supporting tissues, not more than a class I furcation, probe depths up to 6 mm, attachment loss up to 4 mm, could have mobility and radiographic evidence of bone loss
What is slight to moderate loss of support
200
Bacteria usually involved AA, Capnocytophaga. Functional defects in PMS or Monocytes. Onset 12 to age 20, teeth affected perm. 1st molars and incisors. Rate of bone loss 3-4 times CP, unattached gingival plaque, very little inflammation
What is Localized Aggressive P-itis (LAP)
200
Significant PDD beyond what would be expected for the level or plaque and calculus should suggest "something else"
What is P-itis as a manifestation of Systemic Disease
200
HIV/AIDS associated, large areas of necrosis, spontaneous bleeding and punched out papilla, pain and bad breath
What is NUP
200
Vibrational tooth movement under occlusal function (functional mobility)
What is Fremitus
200
Combinations of erythema, edema, BOP, and/or suppuration, loss off > 1/3 supporting tissues, furcation involvement exceeding class I (if present) Probe depths exceeding 6mm with attachment loss of more than 4 mm
What is Advanced loss of periodontal support
300
Usually less that 30 years old, may be uncontrollable. AA, P. Gingivalis AND abnormal neutrophil function.
What is Generalized Aggressive P-itis (GAP)
300
Cardiovascular Disease, PreTerm, Low Birth weight babies, Bacterial pneumonia, NIDDM control
What is P-itis as a risk factor for systemic diseases.
300
AIDS associated as well as Non-AIDs patients, soft tissue necrosis with punched out papilla and sequestration of bone fragments!
What is NUG
300
The most retruded, uppermost and midmost position of the mandibular condyle in relation to the glenoid fossa.
What is Centric relation
300
Pathological detachment of the gingival collagen fibers from the root surface accompanied by apical migration of the junctional epithelium along the root surface.
What is Connective Tissue Attachment loss
400
Onset ages 20-30, fast/generalized pattern of progression, may cease suddenly. P. Gingivitis, P. Intermedia, E. Corodens & Campylobacter recta (yikes!) Treat with tetracycline
What is Rapidly Progressive P-itis (RPP)
400
Onset before age 11, localized or generalized, primary or permanent dentition, associated with Papillon-LeFevre, plaque associated, t-cell defect.
What is What is Prepubertal P-Itis (PPP)
400
Bacteria associated with NUP
What is P. Intermedia & Spirochetes
400
Prevents other teeth from making simultaneous contact
What is occlusal prematurity
400
Attachment loss of 1mm per year (could vary) with episodic busts of activity at different sites, slow bone loss
What is chronic P-itis (exacerbation and remission)
500
Most common form of P-itis onset starting at or later than 35 years, associated with biofilm and host response, most commonly affected teeth are molars and lower anteriors. Gingiva: thick, fibrotic
What is chronic P-itis
500
Associated with local predisposing factors. Modified by systemic dz such as diabetes, stress and tobacco use.
What is Chronic P-itis
500
Anti-rejection medication that causes gingival hyperplasia
What is Cyclosporine
500
Interocclusal space at rest
What is freeway space
500
Progression of gingival inflammation into deeper structures & bone crest. Slight/Crestal bone loss of 1-2mm of (CAL), No mobility, no furcation.
What is Type II Slight (Mild) P-itis