Cardiac Medications
Pain Management
Toxicology
IV Therapy
Misc.
100

The specific drug that prevents clotting by inhibiting Vitamin K

Warfarin (Coumadin)

100

“The perception of pain, resulting from neuralprocessing of pain stimuli"

Nociception
100

Also known as a "toxic fingerprint"

Toxidrome

100

10gtts/mL is this kind of drip set

Macro Drip Set

100

A drug that blocks the effect of another drug

Antagonist

200

The classification of a drug that can cause digitalis effect/toxicity

Cardiac Gylcoside

200

The three medications that can be given as per the ALS PCP Analgesia Medical Directive

Acetaminophen, Ibuprofen, Ketorolac

200

These 3 neurotransmitters are all broken down by monoamine oxidase (MAO)

Norepinephrine, Serotonin, Dopamine

200

Hypotonic solutions cause blood cells to do this

Swell

200

In cholinergic overdoses, this enzyme is blocked from breaking down ACh

Acetylcholinesterase

300

Along with ADP, this component is key to the process of initiating platelet aggregation

Thromboxane (TxA2)

300

The four processes involved in nociception

1. Transduction

2. Transmission

3. Modulation

4. Perception

300

Characterized by a thiamine deficiency and the triad of: an ataxic gait, confusion and abnormal eye movements

Wernicke's Encephalopathy

300

The ALS PCS "Indication" for the intravenous medical directive

The actual or potential need for intravenous medication OR fluid therapy

300

The two types of Cholinergic Receptors

Muscarinic and Nicotinic

400

This type of drug acts on the distal tubules and blocks chloride pumps. (Give the name of the drug as well).

Thiazide Diuretic (HCTZ)

400

This describes the use of agents that have differing onset of action or duration of action to maximize pain relief, as well as to minimize episodes of breakthrough pain

Rational Polypharmacy

400

Na+ and K+ channel blockages can occur in this type of overdose, resulting in prolonged QRS (arrhythmias)

Tricyclic Antidepressants Overdose

400

The TKVO rate for an adult patient

30 to 60 mL/hr

400

These two physiological events occur during an ASA overdose, name in order of occurrence

Respiratory Alkalosis and Metabolic Acidosis

500

The FULL process of the RAAS to increase BP...

Drop in BP => Renin produced by JG Cells => Converts Angiotensinogen from Liver to Angiotensin I => ACE from Lungs converts Angiotensin I to Angiotensin II => Angiotensin II causes vasconstriction of arterioles and hits receptors on adrenal cortex => aldosterone released and promotes increased Na+ and H2O reabsorption => Increase BP

500

Transduction begins when nociceptors of these specific fibres (of afferent neurons) respond to noxious stimuli (provide example of noxious stimuli as well).

"C" and "A-delta" fibres. (Mechanical - Pressure/Thermal - Burn/Chemical - Toxin)

500

Alcohol Dehydrogenase converts Ethyl Alcohol into this and Methyl Alcohol into this. 

H20 + CO2


Formic Acid

500

The per minute drip rate for a patient with an IV running with a macrodrip (10 gtts/mL) set. The patient’s chart indicates that he is to be given 2000 cc of normal saline in 8 hours (to two decimal spots)

41.66 gtts/min

500

The amount of mL you would push for a patient with an order of 2 mg per kg of Norepinephrine I.V. is to be administered. There is pre-mixed syringe of (400mg/10ml) and the patient weights 110 lbs (to one decimal spot)

2.5mL