Etiology/Pathogenesis
The primary virulence factor that allows periodontal pathogens to evade host defense
Endotoxins (lipopolysaccharides)
Interdental Cal of 1-2mm indicates
Stage I
The two main modifiers of Grade
Smoking and diabetes
CAL is measured from
CEJ-base of pocket
A through and through furcation that is not clinically visible is classified as
Class III due to soft tissue, Class IV through and through as tissue is not there
The bacteria most strongly associated with chronic periodontitis
P. Gingivalis
Radiographic bone loss of 15-33% of root length
Stage II
Bone loss divided by age is used to calculate
Grade
6mm pocket with 3mm of recession equals what for CAL
9mm
Which cell type is primarily responsible for collagen production in the periodontal ligament
Fibroblasts
The host response (not bacteria directly) is responsible for most periodontal tissue destruction
Inflammatory Immune Response
Vertical bone loss and moderate ridge defects indicate
Stage III
Heavy bone loss with excellent plaque control suggests
Low current progression rate
Bleeding on probing without attachment loss indicates
Gingivitis
What is a modifying factor for periodontal disease rather than a primary etiologic factor
Occlusal trauma-can modify disease progression
The junctional epithelium migrates apically as a result of
Loss of connective tissue attachment
Loss of posterior support with bite collapse indicates
Stage IV
Mild bone loss with high-risk factors indicates
Grade C
Best clinical indicator of periodontal disease history
Clinical attachment loss
Which cell is responsible for the destruction of alveolar bone in periodontitis
Osteoclast
Why two patients with similar plaque levels may have different disease severity
Host susceptibility
Which findings increases complexity but not severity
Furcation involvement
A non-smoker with uncontrolled diabetes is classified as
at least a Grade B
What clinical finding alone can't diagnose periodontitis
BOP
What is the earliest radiographic sign of disease
Widening of the PDL space