Empty My Trash
Activated Charcoal
Adsorbs toxic substances from the GI tract.
Oxytocin
Increases uterine contractions
Fentanyl
Binds to opiate receptors producing analgesia
Lorazepam
Depresses all levels of the central nervous system (CNS)
Naloxone
Competes and displaces narcotics at the narcotic receptor sites
Inhibits platelet aggregation, has antipyretic and analgesic properties
Dextrose
Increases serum glucose levels. Short-term osmotic diuresis.
Albuterol
Selective beta-2 agonist -Smooth muscle relaxation in the bronchial tree and peripheral vasculature.
Atropine Sulfate
Blocks acetylcholine receptors, increases heart rate, decreases GI secretions
Nitroglycerin
Dilates coronary arteries and vasculature, decreases myocardial oxygen demand
Lidocaine
Cardiac: Suppresses automaticity of conduction tissue by increasing electrical stimulation threshold of ventricle.Local anesthetic: Inhibits transport of ions across the neuronal membrane, blocking conduction of normal nerve impulses.
Diazepam
Potentiates effects of inhibitory neurotransmitters. Raises seizure threshold, induces amnesia and sedation.
Epinephrine 1:10000
Cardiac stimulation and constriction of the vasculature. Stimulates alpha, beta1, and beta2adrenergic receptors resulting in relaxation of smooth muscle of the bronchial tree.
Midazolam
Depresses all levels of Central Nervous System (CNS) function and provides anterograde amnestic effects.
Adenosine
Slows conduction through the AV node; can interrupt reentrant AV nodal pathways
Amiodarone
Blocks sodium, potassium, and calcium channels; prolongs the action potential and repolarization; decreases AV conduction and SA node function.
Diltiazem Hydrochloride
Blocks calcium ion influx during depolarization of cardiac and vascular smooth muscles, slows conduction thru the AV node, decreases rate of ventricular response, decreases myocardial oxygen demand, causes vasodilation.
Diphenhydramine
Blocks H1 histamine receptors in the respiratory tract, blood vessels, and GI smooth muscles; decreases motion sickness. Reverses extrapyramidal reactions.
Morphine Sulfate
Binds to opioid receptors, displacing pain reception. Depresses brainstem respiratory centers. Increases peripheral venous capacitance and decreases venous return.
Blocks the action of acetylcholine at parasympathetic sites in bronchial smooth muscle resulting in bronchodilation.
Epinephrine 1:1000
Stimulates alpha, beta1, and beta2adrenergic receptors resulting in relaxation of smooth muscle of the bronchial tree. Cardiac stimulation and constriction of the vasculature.
Magnesium Sulfate
Magnesium is necessary for the maintaining of serum potassium and calcium levels due to its effect on the renal tubule.
In the heart, it acts as a calcium channel blocker.
Glucagon
Increases blood glucose level by stimulating glycogenolysis. Unknown mechanism of stabilizing cardiac rhythm in beta-blocker overdose. Decreases GI motility and secretions. Minimal positive inotropic and chronotropic response.
Promethazine
H-1 receptor antagonist; blocks action of histamine; sedative, anti-motion, antiemetic, and anticholinergic activity. Potentiates effects of narcotics to induce analgesia.
Dopamine
Immediate metabolic precursor to norepinephrine. Positive inotropic and chronotropic effects. Constricts systemic vasculature, increasing BP and preload. Increases myocardial contractility and stroke volume.